Shaul G. Massry

Research Interests

Organs dysfunction in uremia, phosphate depletion and diabetes mellitus. All these clinical entities have elevated concentration of intracellular calcium. This derangement is responsible in large part for the disturbances in cell function. This finding has permitted us to develop a medical mean to ameliorate and/or reverse organs dysfunction despite the persistence of the disease entity. The use of calcium channel blockers corrects the elevation in the intracellular calcium concentration and as a result ameliorates the complications of these diseases.

Clinical Interests

Internal Medicine
Nephrology
Hypertension
Parathyroid disease
Calcium phosphorous and magnesium
disorders
Renal stones

Degrees

Hebrew University, Hadassah Medical Scho, MD, 1955

Internships

Beilinson Medical Center, Israel, 1954 - 1955

Residencies

Beilinson Medical Center, Israel - Internal Medicine, 1962 - 1964
Beilinson Medical Center, Israel, Chief Medical Resident, 1964 - 1965

Fellowships

Histadrut Institute of Occupational Health & Environmental Physiology, Beilinson, Israel, 1961 - 1962
Georgetown University Hospital, Washington, D.C. - Renal & Electrolyte Division, 1965 - 1966

MEMBERSHIPS & AFFILIATIONS

Memberships

American Society of Nephrology
Western Society of Clinical research
National Kidney Foundation
American Society of Clinical Investigation
Association of American Physicians
American Physiological Society
Endocrine Society
International Society of Nephrology

Clinical Affiliation

USC Ambulatory Health Center
LAC+USC Medical Center
USC Healthcare Consultation Center
USC University Hospital

Bogin E, Massry SG, Harary I: Effect of parathyroid hormone on rat heart cells. J clin Invest. 67:1215-1227,1981

Bogin E, Massry SG, Levi J, Djaldeti M, Bristol G, Smith J. Effect of parathyroid hormone on osmotic fragility of human erythrocytes. J Clin Invest. 69:1017-1025, 1982

Massry SG: Toxic effect of parathyroid hormone in uremia. Seminars Nephrol. 3:306-328, 1983

Massry SG, Klin M, Tian Z, Kedes J, Smogorzweski L&M: Impaired agonist included calcium signaling in hepatocytes from chronic renal failure rate. Kidner Internat. 48;1324-1331, 1995